1<!DOCTYPE html> 2<html lang="en"> 3 <head> 4 <meta http-equiv="Content-Type" content="text/html; charset=utf-8" /> 5 <meta http-equiv="Content-Language" content="en"/> 6 <meta name="viewport" content="width=device-width, initial-scale=1"> 7 8 <title>Gene Expression of LDLR, APOB, APOE, and LPL in Experimentally Induced Diabetes and Dyslipidemia in Rats</title> 9 10 <!-- favicon --> 11 <link rel="shortcut icon" type="image/ico" href="./data/vers/coversheet/favicon.ico" /> 12 13 <!-- user defined metatags --> 14 <meta name="keywords" content="diabetes,Dyslipidemia,gene expression,LDLR,LPL" /> 15 <meta name="description" content="Diabetes is the predominant metabolic condition in humans and animals, particularly in pets; it frequently coexists with many other metabolic abnormalities involving carbohydrates, proteins, and lipids. This study was conducted to investigate the correlation between experimentally generated diabetes and dyslipidemia concerning the gene expression of low-density lipoprotein receptor (LDLR), apolipoprotein-B (APOB), apolipoprotein-E (APOE), and lipoprotein lipase (LPL) in adipose tissue. Sixty-four adult male albino rats were divided into four groups: the first group (control group), receiving a standard diet, while the second group became the dyslipidemia group, which was fed accordingly high-fat diet (HFD), the third group was the diabetes and dyslipidemia group or mixed group fed HFD. Taking a single dosage of 50 mg/kg of streptozotocin (STZ) intraperitoneally, the fourth group became the diabetic group. fed a standard diet and using a single-dose of 50 mg/kg STZ. Results demonstrated that diabetes and dyslipidemia cause significant changes in lipid profile, insulin resistance, and body weight parameters. Dyslipidemia exhibited a significant elevation in total cholesterol and triglycerides relative to the control group. STZ caused a notable elevation in glucose levels and a reduction in insulin levels, those changes resulted in upregulation or downregulation in gene expression of LPL, LDLR, APOB, and APOE. LPL gene expression is downregulated except in the diabetes group, which is slightly upregulated. LDLR gene expression significantly increased in dyslipidemia. It could be concluded that the coexistence of diabetes and dyslipidemia worsens most biochemical markers and gene expression, appearing to be more synergistically harmful than either condition alone." /> 16 <meta name="title" content="Gene Expression of LDLR, APOB, APOE, and LPL in Experimentally Induced Diabetes and Dyslipidemia in Rats" /> 17 <meta name="googlebot" content="NOODP" /> 18 <meta name="citation_title" content="Gene Expression of LDLR, APOB, APOE, and LPL in Experimentally Induced Diabetes and Dyslipidemia in Rats" /> 19 <meta name="citation_author" content="Mahmood, Harikar Salih" /> 20 <meta name="citation_author_institution" content="Dept. of Physiology, Biochemistry, and Pharmacology, College of Veterinary Medicine, University of Mosul" /> 21 <meta name="citation_author" content="Mustafa, Nashaat Ghalib" /> 22 <meta name="citation_author_institution" content="Alkendey" /> 23 <meta name="citation_abstract" content="Diabetes is the predominant metabolic condition in humans and animals, particularly in pets; it frequently coexists with many other metabolic abnormalities involving carbohydrates, proteins, and lipids. This study was conducted to investigate the correlation between experimentally generated diabetes and dyslipidemia concerning the gene expression of low-density lipoprotein receptor (LDLR), apolipoprotein-B (APOB), apolipoprotein-E (APOE), and lipoprotein lipase (LPL) in adipose tissue. Sixty-four adult male albino rats were divided into four groups: the first group (control group), receiving a standard diet, while the second group became the dyslipidemia group, which was fed accordingly high-fat diet (HFD), the third group was the diabetes and dyslipidemia group or mixed group fed HFD. Taking a single dosage of 50 mg/kg of streptozotocin (STZ) intraperitoneally, the fourth group became the diabetic group. fed a standard diet and using a single-dose of 50 mg/kg STZ. Results demonstrated that diabetes and dyslipidemia cause significant changes in lipid profile, insulin resistance, and body weight parameters. Dyslipidemia exhibited a significant elevation in total cholesterol and triglycerides relative to the control group. STZ caused a notable elevation in glucose levels and a reduction in insulin levels, those changes resulted in upregulation or downregulation in gene expression of LPL, LDLR, APOB, and APOE. LPL gene expression is downregulated except in the diabetes group, which is slightly upregulated. LDLR gene expression significantly increased in dyslipidemia. It could be concluded that the coexistence of diabetes and dyslipidemia worsens most biochemical markers and gene expression, appearing to be more synergistically harmful than either condition alone." /> 24 <meta name="citation_id" content="190465" /> 25 <meta name="citation_publication_date" content="2026/07/05" /> 26 <meta name="citation_date" content="2026-07-05" /> 27 <meta name="citation_journal_title" content="Iraqi Journal of Veterinary Scie
27nces" /> 28 <meta name="citation_issn" content="1607-3894" /> 29 <meta name="citation_volume" content="40" /> 30 <meta name="citation_issue" content="3" /> 31 <meta name="citation_firstpage" content="449" /> 32 <meta name="citation_lastpage" content="456" /> 33 <meta name="citation_publisher" content="College of Veterinary Medicine / University of Mosul" /> 34 <meta name="citation_doi" content="10.33899/ijvs.2025.159710.4269" /> 35 <meta name="DC.Identifier" content="10.33899/ijvs.2025.159710.4269" /> 36 <meta name="citation_abstract_html_url" content="https://www.vetmedmosul.com/article_190465.html" /> 37 <meta name="citation_pdf_url" content="https://www.vetmedmosul.com/article_190465_eef340b0309c6cf38def21bc617753e1.pdf" /> 38 <meta name="DC.Title" content="Gene Expression of LDLR, APOB, APOE, and LPL in Experimentally Induced Diabetes and Dyslipidemia in Rats" /> 39 <meta name="DC.Source" content="Iraqi Journal of Veterinary Sciences" /> 40 <meta name="DC.Date" content="05/07/2026" /> 41 <meta name="DC.Date.issued" content="2026-07-05" /> 42 <meta name="DC.Format" content="application/pdf" /> 43 <meta name="DC.Contributor" content="Mahmood, Harikar Salih" /> 44 <meta name="DC.Contributor" content="Mustafa, Nashaat Ghalib" /> 45 <meta name="og:title" content="Gene Expression of LDLR, APOB, APOE, and LPL in Experimentally Induced Diabetes and Dyslipidemia in Rats" /> 46 <meta name="og:description" content="Diabetes is the predominant metabolic condition in humans and animals, particularly in pets; it frequently coexists with many other metabolic abnormalities involving carbohydrates, proteins, and lipids. This study was conducted to investigate the correlation between experimentally generated diabetes and dyslipidemia concerning the gene expression of low-density lipoprotein receptor (LDLR), apolipoprotein-B (APOB), apolipoprotein-E (APOE), and lipoprotein lipase (LPL) in adipose tissue. Sixty-four adult male albino rats were divided into four groups: the first group (control group), receiving a standard diet, while the second group became the dyslipidemia group, which was fed accordingly high-fat diet (HFD), the third group was the diabetes and dyslipidemia group or mixed group fed HFD. Taking a single dosage of 50 mg/kg of streptozotocin (STZ) intraperitoneally, the fourth group became the diabetic group. fed a standard diet and using a single-dose of 50 mg/kg STZ. Results demonstrated that diabetes and dyslipidemia cause significant changes in lipid profile, insulin resistance, and body weight parameters. Dyslipidemia exhibited a significant elevation in total cholesterol and triglycerides relative to the control group. STZ caused a notable elevation in glucose levels and a reduction in insulin levels, those changes resulted in upregulation or downregulation in gene expression of LPL, LDLR, APOB, and APOE. LPL gene expression is downregulated except in the diabetes group, which is slightly upregulated. LDLR gene expression significantly increased in dyslipidemia. It could be concluded that the coexistence of diabetes and dyslipidemia worsens most biochemical markers and gene expression, appearing to be more synergistically harmful than either condition alone." /> 47 <meta name="og:url" content="https://www.vetmedmosul.com/article_190465.html" /> 48 49 <!-- user defined metatags--> 50 <meta name="google-site-verification" content="uznRJhypVtJA5tHKH2vwFYpw1Nh1hmzwdYllU9au8jw" /> 51 <!-- Feed--> 52 <link rel="alternate" type="application/rss+xml" title="RSS feed" href="./ju.rss" /> 53 54 55 56 <!-- CSS --> 57 <link href="./themes/base/front/assets/plugins/bootstrap/css/bootstrap.min.css?v=0.02" rel="stylesheet" type="text/css" /> 58 <link rel="stylesheet" media="screen" href="./themes/theme7/front/assets/css/jquery-ui.min.css" > 59 60 <link href=" ./themes/base/front/assets/css/gfonts-Georgia.css" rel="stylesheet" type="text/css" /> 61 62 <link rel="stylesheet" href="./themes/theme7/front/assets/css/fontawesome-all.min.css"> 63 <link rel="stylesheet" href="./themes/theme7/front/assets/css/select2.min.css" type="text/css" /> 64 <link rel="stylesheet" href="./themes/theme7/front/assets/css/pretty-checkbox.min.css" type="text/css" /> 65 <link rel="stylesheet" href="./themes/theme7/front/assets/css/bootstrap-dialog.min.css" type="text/css" /> 66 <link rel="stylesheet" href="./themes/theme7/front/assets/css/jquery.minipreview.css" type="text/css" /> 67 <link rel="stylesheet" href="./themes/theme7/front/assets/css/cards-style.css" type="text/css" /> 68 <link rel="stylesheet" href="./themes/theme7/front/assets/css/dataTables.bootstrap.min.css" type="text/css" /> 69 <link rel="stylesheet" href="./themes/theme7/front/assets/css/datepicker.css" type="text/css" /> 70 <link rel="stylesheet" href="./themes/base/front/assets/css/academicons.min.css" type="text/css" /> 71 <link rel="stylesheet" href="./themes/base/front/assets/css/social-icon-font.css" type="text/css" /> 72 <link rel="stylesheet" href="./themes/base/front/assets/css/font-awesome.min.css" type="text/css" /> 73 74 <link rel="stylesheet" href="./themes/theme7/front/assets/css/main.css?v=1.6" type="text/css" /> 75 <link rel="stylesheet" href="./themes/theme7/front/assets/css/color.css" type="text/css" /> 76 77 <link href="./inc/css/cstm.css" rel="stylesheet" type="text/css" /> 78 <link href="./data/vers/coversheet/stl_front.css?v=0.88" rel="stylesheet" type="text/css" /> 79 <link href="./data/vers/coversheet/stl.css" rel="stylesheet" type="text/css" /> 80
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Process</a> 424 </label> 425 </div> 426 427 428</div> </div> 429 <div class="col-lg-9 col-md-8 col-sm-12 col-xs-12"> 430 431 <div class="row"> 432 433 <div class="col-lg-9 col-md-8 col-sm-12 col-xs-12"> 434 435 436 <section id="contentList" class="articleInfo"> 437 438 <div class="page-header"> 439 <h1 class="ltr"><a href="./article_190465_eef340b0309c6cf38def21bc617753e1.pdf" target="_blank">Gene Expression of LDLR, APOB, APOE, and LPL in Experimentally Induced Diabetes and Dyslipidemia in Rats</a></h1> 440 </div> 441 442 443 444 445 446 <ul class="authors"> 447 448 <p class="padding-0" style="margin:12px -2px 0 -2px"><strong>Authors</strong></p> 449 450 <ul class="list-inline list-inline-seprator margin-bottom-6 ltr"> 451 <li class="padding-3"> 452 <a href="./?_action=article&au=478364&_au=Harikar+Salih+Mahmood">Harikar Salih Mahmood</a> 453 454 455 <sup><a class="inline-block text-green" href="https://orcid.org/0009-0000-1279-3640" data-toggle="tooltip" data-placement="bottom" title="OrcID: 0009-0000-1279-3640" target="_blank"><i class="ai ai-orcid size-16" ></i></a></sup> 456 457 <sup><a class="inline-block" href="#aff1" >1</a></sup> </li> 458 <li class="padding-3"> 459 <a href="./?_action=article&au=469656&_au=Nashaat+Ghalib+Mustafa">Nashaat Ghalib Mustafa</a> 460 461 <sup><a class="inline-block" href="/cdn-cgi/l/email-protection#402e213328212134777400352f2d2f33352c6e2524356e2931" data-toggle="tooltip" data-placement="bottom" title="Email to Corresponding Author"><i class="fa fa-envelope-o" ></i></a></sup> 462 463 <sup><a class="inline-block text-green" href="https://orcid.org/0000-0002-8351-1230" data-toggle="tooltip" data-placement="bottom" title="OrcID: 0000-0002-8351-1230" target="_blank"><i class="ai ai-orcid size-16" ></i></a></sup> 464 465 <sup><a class="inline-block" href="#aff2" >2</a></sup> </li> 466 </ul> 467 468 <p class="margin-bottom-3 ltr" id="aff1"> 469 <sup>1</sup> 470 Dept. of Physiology, Biochemistry, and Pharmacology, College of Veterinary Medicine, University of Mosul </p> 471 <p class="margin-bottom-3 ltr" id="aff2"> 472 <sup>2</sup> 473 Alkendey </p> 474 475 476 </ul> 477 478 479 480 <div class="cite" style="position:relative"> 481 482 <em> 483 484 </em>, 485 486 <div class="margin-bottom-3"> 487 <em> </em> 488 </div> 489 490 <em><p class="margin-bottom-3">Document Type : Research Paper</p></em> 491 492 493 <div class="margin-bottom-3 ltr" id="ar_doi"><i class="ai ai-doi size-25 text-orange"></i> <span dir="ltr">
493<a href="https://doi.org/10.33899/ijvs.2025.159710.4269">10.33899/ijvs.2025.159710.4269</a></span></div> 494 495 496 497 498 499 </div> 500 501 <div class="articleData"> 502 <ul class="nav nav-tabs"> 503 <li class="active"><a data-toggle="tab" href="#articleTab">Article Information <i class="fa fa-book"></i></a></li> 504 505 506 507 <li><a data-toggle="tab" href="#downloadTab">Download <i class="fa fa-cloud-download" aria-hidden="true"></i></a></li> 508 509 510 511 512 513 514 515 516 <li><a data-toggle="tab" href="#citeTab">Export Citation <i class="fa fa-quote-left"></i></a></li> 517 518 519 <li><a data-toggle="tab" href="#statasticsTab">Statistics <i class="fa fa-chart-bar"></i></a></li> 520 521 522 <li><a data-toggle="tab" href="#shareTab">Share <i class="fa fa-share-alt"></i></a></li> 523 </ul> 524 525 <div class="tab-content"> 526 527 <div id="articleTab" class="tab-pane fade in active"> 528 529 <p class="padding-0" style="margin:12px -2px 0 -2px"><strong>Abstract</strong></p> 530 <p class="justify_txt ltr">Diabetes is the predominant metabolic condition in humans and animals, particularly in pets; it frequently coexists with many other metabolic abnormalities involving carbohydrates, proteins, and lipids. This study was conducted to investigate the correlation between experimentally generated diabetes and dyslipidemia concerning the gene expression of low-density lipoprotein receptor (LDLR), apolipoprotein-B (APOB), apolipoprotein-E (APOE), and lipoprotein lipase (LPL) in adipose tissue. Sixty-four adult male albino rats were divided into four groups: the first group (control group), receiving a standard diet, while the second group became the dyslipidemia group, which was fed accordingly high-fat diet (HFD), the third group was the diabetes and dyslipidemia group or mixed group fed HFD. Taking a single dosage of 50 mg/kg of streptozotocin (STZ) intraperitoneally, the fourth group became the diabetic group. fed a standard diet and using a single-dose of 50 mg/kg STZ. Results demonstrated that diabetes and dyslipidemia cause significant changes in lipid profile, insulin resistance, and body weight parameters. Dyslipidemia exhibited a significant elevation in total cholesterol and triglycerides relative to the control group. STZ caused a notable elevation in glucose levels and a reduction in insulin levels, those changes resulted in upregulation or downregulation in gene expression of LPL, LDLR, APOB, and APOE. LPL gene expression is downregulated except in the diabetes group, which is slightly upregulated. LDLR gene expression significantly increased in dyslipidemia. It could be concluded that the coexistence of diabetes and dyslipidemia worsens most biochemical markers and gene expression, appearing to be more synergistically harmful than either condition alone.</p> 531 532 533 <div class="kw"> 534 535 <p class="padding-0" style="margin:12px -2px 0 -2px"><strong>Keywords</strong></p> 536 537 <ul class="block list-inline list-inline-seprator margin-bottom-6 ltr"> 538 <li class="padding-3"> 539 <a href="./?_action=article&kw=49438&_kw=Diabetes" >Diabetes</a> 540 </li> 541 <li class="padding-3"> 542 <a href="./?_action=article&kw=31467&_kw=Dyslipidemia" >Dyslipidemia</a> 543 </li> 544 <li class="padding-3"> 545 <a href="./?_action=article&kw=69267&_kw=Gene+Expression" >Gene Expression</a> 546 </li> 547 <li class="padding-3"> 548 <a href="./?_action=article&kw=302924&_kw=LDLR" >LDLR</a> 549 </li> 550 <li class="padding-3"> 551 <a href="./?_action=article&kw=308103&_kw=LPL" >LPL</a> 552 </li> 553 </ul> 554 555 </div> 556 557 558 559 560 <div class="kw"> 561 <p class="padding-0" style="margin:12px -2px 0 -2px"><strong>Main Subjects</strong></p> 562 563 <ul class="block list-inline list-inline-seprator margin-bottom-6"> 564 <li class="padding-3"> 565 <a href="./?_action=article&sb=148&_sb=Veterinary+Biochemistry" >Veterinary Biochemistry</a> 566 </li> 567 </ul> 568 </div> 569 570 571 572 573 </div> 574 575 576 577 578 <div id="downloadTab" class="tab-pane fade"> 579 <div class="galleys"> 580 581 582 <!-- Download Files --> 583 <div class="sidebar_block wc"> 584 <div class="content"> 585 <div class="side-nav" > 586 <ul class="list-group list-group-bordered list-group-noicon" style="display:block !important;max-height:9999px"> 587 <li class="list-group-item" ><a href="./?_action=xml&article=190465" target="_blank" class="pdf_link" target="_blank"><i class="fa fa-file-code-o text-orange" ></i> XML</a></li> 588 <li class="list-group-item" ><a href="./article_190465_eef340b0309c6cf38def21bc617753e1.pdf" target="_blank" class="pdf_link" target="_blank"><i class="fa fa-file-pdf-o text-red" ></i> PDF <span dir="ltr" class="badge badge-light">583.52 K</span></a></li> 589 590 591 </ul> 592 </div> 593 </div> 594 </div> 595 596 597 </div> 598 </div> 599 600 601 <div id="citeTab" class="tab-pane fade"> 602 <div> 603 <div id="citationOutput" role="region" aria-l
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937Iraqi Journal of Veterinary Sciences</em>, <em>40</em>(3), 449-456. doi: 10.33899/ijvs.2025.159710.4269</p> 938 </div> 939 </div> 940 </div> 941 </div> 942 <div id="cite-mla" class="modal fade" tabindex="-1" role="dialog" aria-labelledby="myModalLabel" aria-hidden="true"> 943 <div class="modal-dialog"> 944 <div class="modal-content"> 945 946 <!-- Modal Header --> 947 <div class="modal-header"> 948 <button type="button" class="close" data-dismiss="modal" aria-label="Close"><span aria-hidden="true">×</span></button> 949 <h4 class="modal-title" id="myModalLabel">MLA</h4> 950 </div> 951 952 <!-- Modal Body --> 953 <div class="modal-body"> 954 <p>Harikar Salih Mahmood; Nashaat Ghalib Mustafa. "Gene Expression of LDLR, APOB, APOE, and LPL in Experimentally Induced Diabetes and Dyslipidemia in Rats". <em>Iraqi Journal of Veterinary Sciences</em>, 40, 3, 2026, 449-456. doi: 10.33899/ijvs.2025.159710.4269</p> 955 </div> 956 </div> 957 </div> 958 </div> 959 <div id="cite-harvard" class="modal fade" tabindex="-1" role="dialog" aria-labelledby="myModalLabel" aria-hidden="true"> 960 <div class="modal-dialog"> 961 <div class="modal-content"> 962 963 <!-- Modal Header --> 964 <div class="modal-header"> 965 <button type="button" class="close" data-dismiss="modal" aria-label="Close"><span aria-hidden="true">×</span></button> 966 <h4 class="modal-title" id="myModalLabel">HARVARD</h4> 967 </div> 968 969 <!-- Modal Body --> 970 <div class="modal-body"> 971 <p>Mahmood, H., Mustafa, N. (2026). 'Gene Expression of LDLR, APOB, APOE, and LPL in Experimentally Induced Diabetes and Dyslipidemia in Rats', <em>Iraqi Journal of Veterinary Sciences</em>, 40(3), pp. 449-456. doi: 10.33899/ijvs.2025.159710.4269</p> 972 </div> 973 </div> 974 </div> 975 </div> 976 <div id="cite-vancouver" class="modal fade" tabindex="-1" role="dialog" aria-labelledby="myModalLabel" aria-hidden="true"> 977 <div class="modal-dialog"> 978 <div class="modal-content"> 979 980 <!-- Modal Header --> 981 <div class="modal-header"> 982 <button type="button" class="close" data-dismiss="modal" aria-label="Close"><span aria-hidden="true">×</span></button> 983 <h4 class="modal-title" id="myModalLabel">VANCOUVER</h4> 984 </div> 985 986 <!-- Modal Body --> 987 <div class="modal-body"> 988 <p>Mahmood, H., Mustafa, N. Gene Expression of LDLR, APOB, APOE, and LPL in Experimentally Induced Diabetes and Dyslipidemia in Rats. <em>Iraqi Journal of Veterinary Sciences</em>, 2026; 40(3): 449-456. doi: 10.33899/ijvs.2025.159710.4269</p> 989 </div> 990 </div> 991 </div> 992 </div> 993 </div> 994</div> 995<!-- End content register --> 996 997 998 999 </div> 1000 <!-- داÛÙ Ø¨Ø§ÙØ§ Ù Ø±Ø¨ÙØ· ب٠1001 main cotente 1002 --> 1003 <div id="infobox" style="height:0;overflow:hidden"></div> 1004 <footer> 1005 <div id="footer" class="container-fluid"> 1006 <div class="overlay"></div> 1007 <div class="row equal widgetList"> 1008 <div class="col-sm-3 col-xs-12"> 1009 <div class="widgetBox"> 1010 <ul> 1011 <li id="fli_home"><a href="./">Home</a></li> 1012 <li id="fli_about"><a href="./journal/about">About Journal</a></li> 1013 <li id="fli_Edb"><a href="./journal/editorial.board">Editorial Board</a></li> 1014 <li id="fli_submit"><a href="./author">Submit Manuscript</a></li> 1015 <li id="fli_contactus"><a href="./journal/contact.us">Contact Us</a></li> 1016 <li id="fli_glossary"><a href="./journal/glossary">Glossary</a></li> 1017 <li id="fli_sitemap"><a href="./sitemap.xml?usr">Sitemap</a></li> 1018 </ul> 1019 </div> 1020 </div> 1021 <div class="col-sm-3 col-xs-12"> 1022 <div class="widgetBox news"> 1023 <!-- Latest News --> 1024 <h4 class="letter-spacing-1">News</h4> 1025 <ul class="footer-posts list-unstyled"> 1026 <li> 1027 <a href="./news?newsCode=321">The IJVS is implementing an academic integrity system ...</a> 1028 <small>2025-10-01</small> 1029 </li> 1030 <li> 1031 <a href="./news?newsCode=287">Article Processing Charges in IJVS</a> 1032 <small>2024-07-10</small> 1033 </li> 1034 <li> 1035 <a href="./news?newsCode=230">IJVS in the first rank during Iraqi Science day</a> 1036 <small>2023-01-30</small> 1037 </li> 1038 </ul> 1039 <!-- /Latest News --> 1040 </div> 1041 </div> 1042 <div class="col-md-3 margin-bottom-20"> 1043 <!-- Footer Note --> 1044 <div class="widgetBox"><p> </p> 1045<p><strong><span style="font-size: medium; font-family: verdana, geneva;">©<span style="font-size: small;"> 2025, College of Veterinary Medicine, University of Mosul</span></span></strong></p> 1046<p><strong><span style="font-size: medium; font-family: verdana, geneva;"><img style="vertical-align: middle;" src="http://vetmedmosul.com/data/vers/news/by.png" alt="" width="200" height="70" /></span></strong></p> 1047<div id="skyroom-extension-is-installed"></div></div> 1048 </div> 1049 <div class="col-sm-3 col-xs-12"> 1050 <div class="widgetBox"> 1051 <h4 class="letter-spacing-1">Newsletter Subscription</h4> 1052 <p>Subscribe to the journal newsletter and receive the latest news and updates</p> 1053 1054 <form class="validate" action="" method="post" data-success="Subscription saved successfully." data-toastr-position="bottom-right"> 1055 <div class="input-group">
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